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Autophagic flux determines cell death and survival in response to Apo2L/TRAIL (dulanermin). TRID (also known as TRAIL-R3) is an antiapoptotic decoy receptor that lacks the cytoplasmic death domain and competes with KILLER/DR5 and DR4 for binding to TRAIL.
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The recoveries of some compounds are equal to or better than the current EPA methods but with much higher sensitivities. Fatty acid composition of the erythrocyte membranes in patients with chronic respiratory diseases (chronic bronchitis, CB, and stable chronic obstructive pulmonary disease, COPD) was studied.